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More on Cancer
As a general rule, every cell in your body is operating under a
system of "feedback control." A control system of this type is
frequently found among living cells. In trying to explain the
complexity of living cells, perhaps t he best approach is to sim ply say
that the thermostat in your home is much like the feedback mechanisms in
cells. Without actually analyzing the components in detail, it is
sufficent to know that parts of each cell normally function together, in
reacting to whatever environment that the cell is normally found in.
Secondly, DNA is the genetic apparatus which controls the functions and
activities of the living cell. That is, the same DNA is active in each
columnar epithelial cell which lines the intestine of any given ind
ividual, as shown above. And as a general rule, it is the "regular
orientation" or "the standardized activity" of the DNA in each of
those cells which enables them to work in a regular, stablized
manner.
WHY ALL THE CONFUSION ABOUT CANCER?
It has been difficult for researchers to understand how a given
tissue has the ability to order, and/or properly restore its own cells.
It is only now being recognized and published, by Neu Facts, Inc., that
normal cells must communicate with each other by way of Electron
Sensitive Membrane Proteins. These "ESM Proteins," in turn, are linked
into the feed-back control systems, at various levels of activity, in
each cell. Since each cell as shown above, is operating under the same
active DNA , then it is also true that the same "ESM Proteins" must
exist along the sides of every one of those cells. Hence, each cell in
any given tissue is normally linked, via its "ESM Proteins," to the
common information content of every other cell. When you put cells
into a regular laboratory culture however, these cell-to-cell
communications are evidentily no longer active or essential. That
entire system fails when each cell begins to function on an
independent basis.
THE DANGER OF A HIGH-FAT DIET
It is easy to see how fats, and especially poly-unsaturated oils
play an important part in all this. They are difficult to digest. The
fats also coat the membrane surface of the intestinal cells. And if ANY
laboratory animal is put on a high fat diet, mouse or rat, it will
spontaneously develop a cancer and it is usually a cancer of the
colon! 1/24/1990 The results of a high fat diet links perfectly to the
issue of the "ESM Proteins. The key factor is the difficulty which
cells experience in degrading fats. As the levels of undigestible fat
climb, it is obvious that they physically limit regular "ESM Protein"
activity. Hence, the functionally connected feedback system of cellular
regulation is increasingly potentiated to fail. It poses a serious
problem. Abnormal growth ocurs if a cell (or cells) can no longer send
out an electronic signal to confirm its presence among the other cells
of a given tissue. When this happens in colon cancers, most
Doctor's know what usually takes place. A small group of unnecessary
cells starts growing first. These form what is known as a polyp.
Assume, for the moment, that the cells normal ESM Protein performance
has been altered by the fats and that the cells normal communications
system has failed in some of the cells on both side of the long arrow
in the drawing above. The other cells start dividing! They are
designed to reproduce but now they are attempting to replace
what is mistakenly perceived to be dead or otherwise missing, neighbor
cells. A glob of fat has won a very tiny battle, but the real war
doesn't stop there. Those cells will continue to grow until the neck of
that same area cells is totally closed off. And as the enclosed cells
continue to grow, they will cause further problems.
If the patient is fortunate, by seeing a Doctor when the first signs of
bleeding occur, the polyps can be surgically removed. Then a small
sample of those cells, from the polyps, are sent to a laboratory. Once
examined under a microscope, if there are no abnormal cells growing
among those cells, the growth will be declared "benign," or harmless.
The Doctor's say, "We caught it just in time." What this means is:
the genetic apparatus of those cells (the DNA) seems to be in good
working order, and a great many cells were in one place where only a
few should have been.
What Makes Cancer Cells Different? The abnormal operation of the
cell's DNA, is the critical factor and once again that issue is linked
to high dietary fats. In January, 1971, M. J. Hill and associates of
St. Mary's Hospital Medical School in London, England, discovered that
certain gut bacteria might play an important part in the emergence of
colon cancers, based on studies on cancer prone populations. In August
of that same year the same researchers reported that one of the same
bacteria that they had suspected to be somehow able to initiate colon
cancers, was also active in breast cancers. That bacteria, reported to
be a "normal component of the human intestinal flora" was
lecithinase-negative clostridia and they break down fats. In the
process, they also produce two very powerfu l "look alike" female
hormones. Furthermore, it is known that the hormone levels in man drop,
due to extreme stress, or when an individual passes the normal
reproductive stages. Evidently, that's when these bacteria really bloom.
Researchers have found that excessive amounts of hormones (estrone
and estradiol), are carried by post-menopausal women with breast
cancer. In addition, other tissues have been found to "behave like
target organs" for these estrogens, such as the uterus, the vagina, and
the pi tuitary. Cancers emerge there and the same bacteria is found
there in limited numbers. Many other reports in the medical literature
note that a hidden relationship exists between clostridium, cancer, and
the hormones which clostridium produce.
How Does It All Fit Together? The initial cancer cell
emerges when the key features of regular feedback control are lost,
masked, or dropped. It may be due to radiation, a cancer causing
chemical, or the transfer of foreign DNA from a bacteria. The cell's
own DNA becomes altered, or infected with the bacteria's foreign DNA.
The bacteria are smaller than a dot in the drawing, but their DNA
causes a significant change in the ESM Protein of the affected cell.
Once the DNA is transferred, normal cell-to-cell communications fail and
the reordered DNA enables the cancer cell to live and operate on the
bacteria's ESM Proteins. The cell's normal ESM Proteins are lost. And
the body's immune system did not detect the ESM Proteins of the
bacteria, so it will not detect the cancer cells either!
It is also important to note these things:
1. Researchers have had
such a problem with cancers because NONE of the cancer cell features
have ever fallen under the ordinary rules of bacteriology and disease.
2. The millions of cells, which make up each cancer, have each unfolded
with the same properties of the first, initial cancer cell.
3. The
altered DNA reorganizes membrane features which obviously lends
themselves to a flexible and unstablized range of activities,
relative to a tissue's ability to order and/or properly restore it's own
numbers.
4. The bacteria involved in each cancer can be easily
identified because they normally exist among the cancer cells which some
of their own DNA, in part, initially produced, and
5. These bacteria
also produce hormone sensitive cancers, such as breast cancers, via
"hormone carriers" which transport or move the offending DNA through the
host's blood stream to the hormone-sensitive cells.
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